The neurologic phenotype in SCA8 BAC expansion although not BAC Regulate lines demonstrated the pathogenicity from the (CTG-CAG)n expansion. Also, the expression of noncoding (CUG)n enlargement ATXN8OS transcripts and the invention of intranuclear polyglutamine inclusions suggested that SCA8 pathogenesis requires toxic achieve-of-purpose mechanisms at each the protein along with the https://silask887dqa1.wikihearsay.com/user